Anatomy & Physiology: The Unity of Form and Function, 7th Edition

Published by McGraw-Hill Education
ISBN 10: 0073403717
ISBN 13: 978-0-07340-371-7

Chapter 19 - The Circulatory System: The Heart - Study Guide - Testing Your Comprehension - Page 742: 3

Answer

Becky's ventricular septal defect (VSD) refers to a hole in the interventricular septum, which is the wall that separates the two ventricles of the heart. In a healthy heart, the left ventricle pumps oxygen-rich blood to the systemic circulation, while the right ventricle pumps oxygen-poor blood to the lungs for oxygenation. Let's consider the effects of Becky's VSD on pulmonary blood pressure, systemic blood pressure, and the long-term changes in the ventricular walls: 1. **Pulmonary Blood Pressure:** The left ventricle normally pumps blood into the systemic circulation, which has a higher pressure requirement to distribute blood throughout the body. Because of the VSD, some of the oxygen-rich blood from the left ventricle could flow back into the right ventricle, mixing with oxygen-poor blood. This might lead to increased blood volume and pressure in the right ventricle. The increased volume and pressure in the right ventricle can potentially result in higher pressure in the pulmonary circulation, as more blood is pushed into the pulmonary arteries. Over time, this could lead to increased pulmonary blood pressure, which might strain the pulmonary arteries and the right side of the heart. 2. **Systemic Blood Pressure:** The mixing of oxygen-rich blood from the left ventricle with oxygen-poor blood from the right ventricle can result in decreased oxygen content in the systemic circulation. This might lead to reduced systemic blood pressure due to the lower oxygen supply to the body's tissues. The body might compensate for this by increasing heart rate and cardiac output to maintain adequate oxygen delivery to the tissues. However, this compensation might not fully counteract the impact of the VSD on systemic blood pressure. 3. **Long-Term Changes in Ventricular Walls:** Over time, the increased workload on the right ventricle due to the VSD might lead to its enlargement and thickening (hypertrophy) as it works harder to push blood against the increased pulmonary pressure. The left ventricle might also undergo changes in response to the increased volume and pressure load. These changes in ventricular walls are adaptations aimed at maintaining cardiac output but can eventually lead to heart function impairment if left untreated. It's important to note that the effects of VSD can vary based on factors such as the size of the defect, its location, and individual variations in anatomy and physiology. Early medical intervention and, in some cases, surgical repair are often recommended to mitigate these effects and prevent long-term complications. Pediatric cardiologists will closely monitor Becky's condition to determine the appropriate treatment plan to ensure her heart functions as effectively as possible.

Work Step by Step

Becky's ventricular septal defect (VSD) refers to a hole in the interventricular septum, which is the wall that separates the two ventricles of the heart. In a healthy heart, the left ventricle pumps oxygen-rich blood to the systemic circulation, while the right ventricle pumps oxygen-poor blood to the lungs for oxygenation. Let's consider the effects of Becky's VSD on pulmonary blood pressure, systemic blood pressure, and the long-term changes in the ventricular walls: 1. **Pulmonary Blood Pressure:** The left ventricle normally pumps blood into the systemic circulation, which has a higher pressure requirement to distribute blood throughout the body. Because of the VSD, some of the oxygen-rich blood from the left ventricle could flow back into the right ventricle, mixing with oxygen-poor blood. This might lead to increased blood volume and pressure in the right ventricle. The increased volume and pressure in the right ventricle can potentially result in higher pressure in the pulmonary circulation, as more blood is pushed into the pulmonary arteries. Over time, this could lead to increased pulmonary blood pressure, which might strain the pulmonary arteries and the right side of the heart. 2. **Systemic Blood Pressure:** The mixing of oxygen-rich blood from the left ventricle with oxygen-poor blood from the right ventricle can result in decreased oxygen content in the systemic circulation. This might lead to reduced systemic blood pressure due to the lower oxygen supply to the body's tissues. The body might compensate for this by increasing heart rate and cardiac output to maintain adequate oxygen delivery to the tissues. However, this compensation might not fully counteract the impact of the VSD on systemic blood pressure. 3. **Long-Term Changes in Ventricular Walls:** Over time, the increased workload on the right ventricle due to the VSD might lead to its enlargement and thickening (hypertrophy) as it works harder to push blood against the increased pulmonary pressure. The left ventricle might also undergo changes in response to the increased volume and pressure load. These changes in ventricular walls are adaptations aimed at maintaining cardiac output but can eventually lead to heart function impairment if left untreated. It's important to note that the effects of VSD can vary based on factors such as the size of the defect, its location, and individual variations in anatomy and physiology. Early medical intervention and, in some cases, surgical repair are often recommended to mitigate these effects and prevent long-term complications. Pediatric cardiologists will closely monitor Becky's condition to determine the appropriate treatment plan to ensure her heart functions as effectively as possible.
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